• 4546 Citations
  • 36 h-Index
19952019
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Fingerprint Dive into the research topics where Alessandro Pecci is active. These topic labels come from the works of this person. Together they form a unique fingerprint.

  • 18 Similar Profiles
Thrombocytopenia Medicine & Life Sciences
Mutation Medicine & Life Sciences
Blood Platelets Medicine & Life Sciences
Nonmuscle Myosin Type IIA Medicine & Life Sciences
Megakaryocytes Medicine & Life Sciences
Myosin Heavy Chains Medicine & Life Sciences
Myelodysplastic Syndromes Medicine & Life Sciences
Deafness Medicine & Life Sciences

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Research Output 1995 2019

  • 4546 Citations
  • 36 h-Index
  • 136 Article
  • 4 Letter
  • 4 Review article

Lemierre Syndrome: Clinical Update and Protocol for a Systematic Review and Individual Patient Data Meta-analysis

Sacco, C., Zane, F., Granziera, S., Holm, K., Creemers-Schild, D., Hotz, M-A., Turpini, E., Valentini, A., Righini, C., Karkos, P. D., Verhamme, P., Di Nisio, M., Konstantinides, S., Pecci, A., Barco, S. & the, O. B. O., 2019, In : Hamostaseologie. 39, 01, p. 76-86 11 p.

Research output: Contribution to journalArticle

Loss-of-function mutations in PTPRJ cause a new form of inherited thrombocytopenia

Marconi, C., DI Buduo, C. A., LeVine, K., Barozzi, S., Faleschini, M., Bozzi, V., Palombo, F., McKinstry, S., Lassandro, G., Giordano, P., Noris, P., Balduini, C. L., Savoia, A., Balduini, A., Pippucci, T., Seri, M., Katsanis, N. & Pecci, A., 2019, In : Blood. 133, 12, p. 1346-1357 12 p.

Research output: Contribution to journalArticle

Platelets
Thrombocytopenia
Blood Platelets
Mutation
Megakaryocytes

Loss-of-function mutations in PTPRJ cause a new form of inherited thrombocytopenia

Marconi, C., Di Buduo, C. A., LeVine, K., Barozzi, S., Faleschini, M., Bozzi, V., Palombo, F., McKinstry, S., Lassandro, G., Giordano, P., Noris, P., Balduini, C. L., Savoia, A., Balduini, A., Pippucci, T., Seri, M., Katsanis, N. & Pecci, A., 2019, In : Blood. 133, 12, p. 1346-1357 12 p.

Research output: Contribution to journalArticle

MYH9-related disease mutations cause abnormal red blood cell morphology through increased myosin-actin binding at the membrane

Smith, A. S., Pal, K., Nowak, R. B., Demenko, A., Zaninetti, C., Da Costa, L., Favier, R., Pecci, A. & Fowler, V. M., Jan 1 2019, In : American Journal of Hematology.

Research output: Contribution to journalArticle

Nonmuscle Myosin Type IIA
Myosins
Actins
Erythrocytes
Mutation