Activated DDR1 increases RS cell survival

Antonino Carbone, Annunziata Gloghini

Research output: Contribution to journalArticle

Abstract

In this issue of Blood, Cader et al show that tumor microenvironment promotes Epstein-Barr virus (EBV)-driven lymphomagenesis in Hodgkin lymphoma by a novel pathway involving latent membrane protein 1 (LMP1) and discoidin domain receptor 1 (DDR1), which is activated by collagen(s) and contributes to the survival of Reed-Sternberg (RS) cells.

Original languageEnglish
Pages (from-to)4152-4154
Number of pages3
JournalBlood
Volume122
Issue number26
DOIs
Publication statusPublished - Dec 19 2013

Fingerprint

Reed-Sternberg Cells
Tumor Microenvironment
Human Herpesvirus 4
Hodgkin Disease
Viruses
Tumors
Cell Survival
Membrane Proteins
Blood
Collagen
Cells
Discoidin Domain Receptor 1
Protein Domains

ASJC Scopus subject areas

  • Hematology
  • Biochemistry
  • Cell Biology
  • Immunology

Cite this

Activated DDR1 increases RS cell survival. / Carbone, Antonino; Gloghini, Annunziata.

In: Blood, Vol. 122, No. 26, 19.12.2013, p. 4152-4154.

Research output: Contribution to journalArticle

Carbone, Antonino ; Gloghini, Annunziata. / Activated DDR1 increases RS cell survival. In: Blood. 2013 ; Vol. 122, No. 26. pp. 4152-4154.
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