Corrigendum: Translation from a DMD exon 5 IRES results in a functional dystrophin isoform that attenuates dystrophinopathy in humans and mice (Nature Medicine (2014))

Nicolas Wein; Adeline Vulin; Maria S. Falzarano; Christina Al Khalili Szigyarto; Baijayanta Maiti; Andrew Findlay; Kristin N. Heller; Mathias Uhlén; Baskar Bakthavachalu; Sonia Messina; Giuseppe Vita; Chiara Passarelli; Francesca Gualandi; Steve D. Wilton; Louise R. Rodino-Klapac; Lin Yang; Diane M. Dunn; Daniel R. Schoenberg; Robert B. Weiss; Michael T. Howard; Alessandra Ferlini; Kevin M. Flanigan

doi:10.1038/nm0415-414b

Corrigendum: Translation from a DMD exon 5 IRES results in a functional dystrophin isoform that attenuates dystrophinopathy in humans and mice (Nature Medicine (2014))

Nicolas Wein, Adeline Vulin, Maria S. Falzarano, Christina Al Khalili Szigyarto, Baijayanta Maiti, Andrew Findlay, Kristin N. Heller, Mathias Uhlén, Baskar Bakthavachalu, Sonia Messina, Giuseppe Vita, Chiara Passarelli, Francesca Gualandi, Steve D. Wilton, Louise R. Rodino-Klapac, Lin Yang, Diane M. Dunn, Daniel R. Schoenberg, Robert B. Weiss, Michael T. HowardAlessandra Ferlini, Kevin M. Flanigan

Ospedale Pediatrico Bambino Gesu

Research output: Contribution to journal › Article › peer-review

Original language	English
Pages (from-to)	414
Number of pages	1
Journal	Nature Medicine
Volume	21
Issue number	4
DOIs	https://doi.org/10.1038/nm0415-414b
Publication status	Published - Apr 1 2015

ASJC Scopus subject areas

Biochemistry, Genetics and Molecular Biology(all)
Medicine(all)

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Wein, N., Vulin, A., Falzarano, M. S., Szigyarto, C. A. K., Maiti, B., Findlay, A., Heller, K. N., Uhlén, M., Bakthavachalu, B., Messina, S., Vita, G., Passarelli, C., Gualandi, F., Wilton, S. D., Rodino-Klapac, L. R., Yang, L., Dunn, D. M., Schoenberg, D. R., Weiss, R. B., ... Flanigan, K. M. (2015). Corrigendum: Translation from a DMD exon 5 IRES results in a functional dystrophin isoform that attenuates dystrophinopathy in humans and mice (Nature Medicine (2014)). Nature Medicine, 21(4), 414. https://doi.org/10.1038/nm0415-414b

Corrigendum : Translation from a DMD exon 5 IRES results in a functional dystrophin isoform that attenuates dystrophinopathy in humans and mice (Nature Medicine (2014)). / Wein, Nicolas; Vulin, Adeline; Falzarano, Maria S.; Szigyarto, Christina Al Khalili; Maiti, Baijayanta; Findlay, Andrew; Heller, Kristin N.; Uhlén, Mathias; Bakthavachalu, Baskar; Messina, Sonia; Vita, Giuseppe; Passarelli, Chiara; Gualandi, Francesca; Wilton, Steve D.; Rodino-Klapac, Louise R.; Yang, Lin; Dunn, Diane M.; Schoenberg, Daniel R.; Weiss, Robert B.; Howard, Michael T.; Ferlini, Alessandra; Flanigan, Kevin M.

In: Nature Medicine, Vol. 21, No. 4, 01.04.2015, p. 414.

Research output: Contribution to journal › Article › peer-review

Wein, N, Vulin, A, Falzarano, MS, Szigyarto, CAK, Maiti, B, Findlay, A, Heller, KN, Uhlén, M, Bakthavachalu, B, Messina, S, Vita, G, Passarelli, C, Gualandi, F, Wilton, SD, Rodino-Klapac, LR, Yang, L, Dunn, DM, Schoenberg, DR, Weiss, RB, Howard, MT, Ferlini, A & Flanigan, KM 2015, 'Corrigendum: Translation from a DMD exon 5 IRES results in a functional dystrophin isoform that attenuates dystrophinopathy in humans and mice (Nature Medicine (2014))', Nature Medicine, vol. 21, no. 4, pp. 414. https://doi.org/10.1038/nm0415-414b

Wein, Nicolas ; Vulin, Adeline ; Falzarano, Maria S. ; Szigyarto, Christina Al Khalili ; Maiti, Baijayanta ; Findlay, Andrew ; Heller, Kristin N. ; Uhlén, Mathias ; Bakthavachalu, Baskar ; Messina, Sonia ; Vita, Giuseppe ; Passarelli, Chiara ; Gualandi, Francesca ; Wilton, Steve D. ; Rodino-Klapac, Louise R. ; Yang, Lin ; Dunn, Diane M. ; Schoenberg, Daniel R. ; Weiss, Robert B. ; Howard, Michael T. ; Ferlini, Alessandra ; Flanigan, Kevin M. / Corrigendum : Translation from a DMD exon 5 IRES results in a functional dystrophin isoform that attenuates dystrophinopathy in humans and mice (Nature Medicine (2014)). In: Nature Medicine. 2015 ; Vol. 21, No. 4. pp. 414.

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Corrigendum: Translation from a DMD exon 5 IRES results in a functional dystrophin isoform that attenuates dystrophinopathy in humans and mice (Nature Medicine (2014))

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