Abstract
We analyzed some particular aspects of the peripheral pathways likely to be involved in the mediation of cardiac pain. The functional characteristics and the reflex function of cardiac sympathetic afferent fibers are discussed in relation to the afferent code transmitting nociception. A modified version of the intensity theory based on the extreme excitation of a spatially restricted population of afferent sympathetic fibers is proposed as the most likely mechanism involved in the genesis of cardiac pain.
| Original language | English |
|---|---|
| Journal | Journal of Cardiovascular Electrophysiology |
| Volume | 2 |
| Issue number | 2 SUPPL. |
| Publication status | Published - 1991 |
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ASJC Scopus subject areas
- Physiology
- Cardiology and Cardiovascular Medicine
Cite this
From cardiac nociception to the brain : The unstable character of angina pectoris. / Lombardi, F.; Malliani, A.
In: Journal of Cardiovascular Electrophysiology, Vol. 2, No. 2 SUPPL., 1991.Research output: Contribution to journal › Article
}
TY - JOUR
T1 - From cardiac nociception to the brain
T2 - The unstable character of angina pectoris
AU - Lombardi, F.
AU - Malliani, A.
PY - 1991
Y1 - 1991
N2 - We analyzed some particular aspects of the peripheral pathways likely to be involved in the mediation of cardiac pain. The functional characteristics and the reflex function of cardiac sympathetic afferent fibers are discussed in relation to the afferent code transmitting nociception. A modified version of the intensity theory based on the extreme excitation of a spatially restricted population of afferent sympathetic fibers is proposed as the most likely mechanism involved in the genesis of cardiac pain.
AB - We analyzed some particular aspects of the peripheral pathways likely to be involved in the mediation of cardiac pain. The functional characteristics and the reflex function of cardiac sympathetic afferent fibers are discussed in relation to the afferent code transmitting nociception. A modified version of the intensity theory based on the extreme excitation of a spatially restricted population of afferent sympathetic fibers is proposed as the most likely mechanism involved in the genesis of cardiac pain.
UR - http://www.scopus.com/inward/record.url?scp=0025731722&partnerID=8YFLogxK
UR - http://www.scopus.com/inward/citedby.url?scp=0025731722&partnerID=8YFLogxK
M3 - Article
AN - SCOPUS:0025731722
VL - 2
JO - Journal of Cardiovascular Electrophysiology
JF - Journal of Cardiovascular Electrophysiology
SN - 1045-3873
IS - 2 SUPPL.
ER -