This work presents a case of primary hypoparathyroidism in an adult in whom the disorder for years ran a course of only convulsive attacks, followed later on by a bilateral cataract. Treatment of the attacks with barbiturates and hydantoin proved inefficacious. During a tonic and clonic state of grand mal a severe hypocalcaemia was found. This settled the question of the diagnosis. The normalization of the calcaemia led to the attacks vanishing and improved the other symptoms of the disease. The antiepileptic drugs were suspended. The development of the EEG is discussed and also the possibility that the chronic antiepileptic treatment had exacerbated the syndrome by a further reduction of the calcaemia by a secondary deficiency of vitamin D.
|Translated title of the contribution||Grand mal seizures due to idiopathic hypoparathyroidism; details of a case|
|Title of host publication||RIV. ITAL. ELETTROENCEFALOGR. NEUROFISIOL.|
|Number of pages||5|
|Publication status||Published - 1979|
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