IPOPARATIROIDISMO IDIOPATICO E CRISI COMIZIALI

Translated title of the contribution: Idiopathic hyperparathyroidism and epileptic seizures

P. Maestripieri, F. Vigevano

Research output: Contribution to journalArticle

1 Citation (Scopus)

Abstract

In a patient with primary hypoparathyroidism the disease ran for years with only generalized convulsions resistant to antiepileptic medication, and was later complicated by bilateral cataracts. Low serum calcium and elevated serum phosphorus, found on occasion of a grand mal status resistant to routine treatment and to intravenous barbiturates, revealed the correct diagnosis. The seizures were suppressed by normalizing the serum calcium levels. The pathophysiology of convulsive seizures during hypocalcemia is discussed, and the possibility is raised that antiepileptic drugs could have contributed to hypocalcemia through a vitamin D deficiency.

Original languageItalian
Pages (from-to)56-66
Number of pages11
JournalRivista di Neurologia
Volume50
Issue number1
Publication statusPublished - 1980

Fingerprint

Hyperparathyroidism
Epilepsy
Seizures
Hypocalcemia
Anticonvulsants
Serum
Calcium
Hypoparathyroidism
Barbiturates
Vitamin D Deficiency
Cataract
Phosphorus
Therapeutics

ASJC Scopus subject areas

  • Clinical Neurology

Cite this

IPOPARATIROIDISMO IDIOPATICO E CRISI COMIZIALI. / Maestripieri, P.; Vigevano, F.

In: Rivista di Neurologia, Vol. 50, No. 1, 1980, p. 56-66.

Research output: Contribution to journalArticle

Maestripieri, P & Vigevano, F 1980, 'IPOPARATIROIDISMO IDIOPATICO E CRISI COMIZIALI', Rivista di Neurologia, vol. 50, no. 1, pp. 56-66.
Maestripieri, P. ; Vigevano, F. / IPOPARATIROIDISMO IDIOPATICO E CRISI COMIZIALI. In: Rivista di Neurologia. 1980 ; Vol. 50, No. 1. pp. 56-66.
@article{662e00eb97f642f788c87b555b638460,
title = "IPOPARATIROIDISMO IDIOPATICO E CRISI COMIZIALI",
abstract = "In a patient with primary hypoparathyroidism the disease ran for years with only generalized convulsions resistant to antiepileptic medication, and was later complicated by bilateral cataracts. Low serum calcium and elevated serum phosphorus, found on occasion of a grand mal status resistant to routine treatment and to intravenous barbiturates, revealed the correct diagnosis. The seizures were suppressed by normalizing the serum calcium levels. The pathophysiology of convulsive seizures during hypocalcemia is discussed, and the possibility is raised that antiepileptic drugs could have contributed to hypocalcemia through a vitamin D deficiency.",
author = "P. Maestripieri and F. Vigevano",
year = "1980",
language = "Italian",
volume = "50",
pages = "56--66",
journal = "Rivista di Neurologia",
issn = "0035-6344",
publisher = "Il Pensiero Scientifico Editore s.r.l.",
number = "1",

}

TY - JOUR

T1 - IPOPARATIROIDISMO IDIOPATICO E CRISI COMIZIALI

AU - Maestripieri, P.

AU - Vigevano, F.

PY - 1980

Y1 - 1980

N2 - In a patient with primary hypoparathyroidism the disease ran for years with only generalized convulsions resistant to antiepileptic medication, and was later complicated by bilateral cataracts. Low serum calcium and elevated serum phosphorus, found on occasion of a grand mal status resistant to routine treatment and to intravenous barbiturates, revealed the correct diagnosis. The seizures were suppressed by normalizing the serum calcium levels. The pathophysiology of convulsive seizures during hypocalcemia is discussed, and the possibility is raised that antiepileptic drugs could have contributed to hypocalcemia through a vitamin D deficiency.

AB - In a patient with primary hypoparathyroidism the disease ran for years with only generalized convulsions resistant to antiepileptic medication, and was later complicated by bilateral cataracts. Low serum calcium and elevated serum phosphorus, found on occasion of a grand mal status resistant to routine treatment and to intravenous barbiturates, revealed the correct diagnosis. The seizures were suppressed by normalizing the serum calcium levels. The pathophysiology of convulsive seizures during hypocalcemia is discussed, and the possibility is raised that antiepileptic drugs could have contributed to hypocalcemia through a vitamin D deficiency.

UR - http://www.scopus.com/inward/record.url?scp=0018851262&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=0018851262&partnerID=8YFLogxK

M3 - Articolo

VL - 50

SP - 56

EP - 66

JO - Rivista di Neurologia

JF - Rivista di Neurologia

SN - 0035-6344

IS - 1

ER -