Abstract
The studies described in this article support a role for immunologic-inflammatory mechanisms in the pathogenesis of atherosclerosis. This immunologic-inflammatory state is evident in many autoimmune diseases, but also in the general population lacking an overt autoimmune disease. The ability to immunomodulate atherosclerosis (currently only experimental) should lead to future research into the mechanisms and treatment of atherosclerosis, the leading cause of death in the Western world.
Original language | English |
---|---|
Pages (from-to) | 355-362 |
Number of pages | 8 |
Journal | Rheumatic Disease Clinics of North America |
Volume | 31 |
Issue number | 2 |
DOIs | |
Publication status | Published - May 2005 |
ASJC Scopus subject areas
- Rheumatology