N1 methylnicotinamide urinary output in multiple sclerosis affected patients

E. Sacchetti, A. C. Altamura, E. Smeraldi, A. Zibetti

Research output: Contribution to journalArticle

Abstract

This brief report develops the ideas of previous work underlying a possible functional disequilibrium of tryptophan metabolism in patients affected by multiple sclerosis (MS) and in animals with experimental allergic encephalomyelitis. The urinary excretion of one of the terminal metabolites of the kynurenine pathway of tryptophan, N1 methylnicotinamide, has been confronted in MS patients and in healthy volunteers in order to show a possible alteration in MS patients. The authors could not find any significant difference in N1 methylnicotinamide urinary output between controls and MS patients.

Original languageEnglish
Pages (from-to)206-209
Number of pages4
JournalActa Vitaminologica et Enzymologica
Volume29
Issue number1-6
Publication statusPublished - 1975

ASJC Scopus subject areas

  • Medicine(all)

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