Reversal of proximal tubular dysfunction by indomethacin

M. Usberti, M. Mileti, C. G. D'Auria, A. Dal Canton, C. Romano, B. Copercini, C. Emanuelli, F. Pecchini

Research output: Contribution to journalArticlepeer-review


This paper reports a patient with salt-losing nephropathy, phosphaturia, glucosuria, hypercalciuria and bicarbonaturia, i.e. urinary abnormalities due to proximal tubular defects. Oral indomethacin caused a complete normalization of urinary alterations. These results show that indomethacin is effective in the treatment of proximal tubular defects and suggest that this therapeutic effect is due to an overall increase in proximal tubular reabsorption.

Original languageEnglish
Pages (from-to)358-360
Number of pages3
Issue number4
Publication statusPublished - 1982

ASJC Scopus subject areas

  • Nephrology


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