Abstract
1. In vagotomized anaesthetized cats with both common carotid arteries occluded, stretch of the thoracic aorta induced reflex increases in arterial blood pressure, heart rate and left ventricular dP/dtmax. Similar responses were obtained in cats with spinal transection at the level of the first cervical nerve roots. 2. The hypothesis is advanced that sympathetic excitatory reflexes may contribute to the maintenance of hypertension through positive feedback.
| Original language | English |
|---|---|
| Journal | Clinical science and molecular medicine. Supplement |
| Volume | 2 |
| Publication status | Published - Jun 1975 |
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ASJC Scopus subject areas
- Medicine(all)
Cite this
Spinal sympathetic reflexes in the cat and the pathogenesis of arterial hypertension. / Malliani, A.; Lombardi, F.; Pagani, M.; Recordati, G.; Schwartz, P. J.
In: Clinical science and molecular medicine. Supplement, Vol. 2, 06.1975.Research output: Contribution to journal › Article
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TY - JOUR
T1 - Spinal sympathetic reflexes in the cat and the pathogenesis of arterial hypertension.
AU - Malliani, A.
AU - Lombardi, F.
AU - Pagani, M.
AU - Recordati, G.
AU - Schwartz, P. J.
PY - 1975/6
Y1 - 1975/6
N2 - 1. In vagotomized anaesthetized cats with both common carotid arteries occluded, stretch of the thoracic aorta induced reflex increases in arterial blood pressure, heart rate and left ventricular dP/dtmax. Similar responses were obtained in cats with spinal transection at the level of the first cervical nerve roots. 2. The hypothesis is advanced that sympathetic excitatory reflexes may contribute to the maintenance of hypertension through positive feedback.
AB - 1. In vagotomized anaesthetized cats with both common carotid arteries occluded, stretch of the thoracic aorta induced reflex increases in arterial blood pressure, heart rate and left ventricular dP/dtmax. Similar responses were obtained in cats with spinal transection at the level of the first cervical nerve roots. 2. The hypothesis is advanced that sympathetic excitatory reflexes may contribute to the maintenance of hypertension through positive feedback.
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UR - http://www.scopus.com/inward/citedby.url?scp=0016511386&partnerID=8YFLogxK
M3 - Article
VL - 2
JO - Clinical science and molecular medicine. Supplement
JF - Clinical science and molecular medicine. Supplement
SN - 0144-4107
ER -