Stage 1 type 3 neovascularization with choroidal dilation unresponsive to anti-VEGF treatment

Stela Vujosevic, Ferdinando Martini, Edoardo Midena

Research output: Contribution to journalArticle

Abstract

Type 3 neovascularization is considered to originate within the retina with subsequent expansion of the neovascular network into the subretinal space. Choroidal circulatory disturbances seem to have a role in Type 3 neovascularization development, but the exact pathophysiology is still under debate. Although eyes with Type 3 neovascularization usually have thinner choroid compared to normal eyes, the increased choroidal thickness associated with Type 3 neovascularization may be a prognostic factor for its recurrence. This case report documents stage 1 Type 3 neovascularization with dilated choroid showing poor response to anti-vascular endothelial growth factor therapy, thus suggesting an active role of choroid in Type 3 lesions.

Original languageEnglish
Pages (from-to)956-959
Number of pages4
JournalOphthalmic Surgery Lasers and Imaging Retina
Volume47
Issue number10
DOIs
Publication statusPublished - Oct 1 2016

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Choroidal Neovascularization
Choroid
Vascular Endothelial Growth Factor A
Dilatation
Retina
Recurrence
Therapeutics

ASJC Scopus subject areas

  • Surgery
  • Ophthalmology

Cite this

Stage 1 type 3 neovascularization with choroidal dilation unresponsive to anti-VEGF treatment. / Vujosevic, Stela; Martini, Ferdinando; Midena, Edoardo.

In: Ophthalmic Surgery Lasers and Imaging Retina, Vol. 47, No. 10, 01.10.2016, p. 956-959.

Research output: Contribution to journalArticle

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