Abstract
Type 3 neovascularization is considered to originate within the retina with subsequent expansion of the neovascular network into the subretinal space. Choroidal circulatory disturbances seem to have a role in Type 3 neovascularization development, but the exact pathophysiology is still under debate. Although eyes with Type 3 neovascularization usually have thinner choroid compared to normal eyes, the increased choroidal thickness associated with Type 3 neovascularization may be a prognostic factor for its recurrence. This case report documents stage 1 Type 3 neovascularization with dilated choroid showing poor response to anti-vascular endothelial growth factor therapy, thus suggesting an active role of choroid in Type 3 lesions.
| Original language | English |
|---|---|
| Pages (from-to) | 956-959 |
| Number of pages | 4 |
| Journal | Ophthalmic Surgery Lasers and Imaging Retina |
| Volume | 47 |
| Issue number | 10 |
| DOIs | |
| Publication status | Published - Oct 1 2016 |
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ASJC Scopus subject areas
- Surgery
- Ophthalmology
Cite this
Stage 1 type 3 neovascularization with choroidal dilation unresponsive to anti-VEGF treatment. / Vujosevic, Stela; Martini, Ferdinando; Midena, Edoardo.
In: Ophthalmic Surgery Lasers and Imaging Retina, Vol. 47, No. 10, 01.10.2016, p. 956-959.Research output: Contribution to journal › Article
}
TY - JOUR
T1 - Stage 1 type 3 neovascularization with choroidal dilation unresponsive to anti-VEGF treatment
AU - Vujosevic, Stela
AU - Martini, Ferdinando
AU - Midena, Edoardo
PY - 2016/10/1
Y1 - 2016/10/1
N2 - Type 3 neovascularization is considered to originate within the retina with subsequent expansion of the neovascular network into the subretinal space. Choroidal circulatory disturbances seem to have a role in Type 3 neovascularization development, but the exact pathophysiology is still under debate. Although eyes with Type 3 neovascularization usually have thinner choroid compared to normal eyes, the increased choroidal thickness associated with Type 3 neovascularization may be a prognostic factor for its recurrence. This case report documents stage 1 Type 3 neovascularization with dilated choroid showing poor response to anti-vascular endothelial growth factor therapy, thus suggesting an active role of choroid in Type 3 lesions.
AB - Type 3 neovascularization is considered to originate within the retina with subsequent expansion of the neovascular network into the subretinal space. Choroidal circulatory disturbances seem to have a role in Type 3 neovascularization development, but the exact pathophysiology is still under debate. Although eyes with Type 3 neovascularization usually have thinner choroid compared to normal eyes, the increased choroidal thickness associated with Type 3 neovascularization may be a prognostic factor for its recurrence. This case report documents stage 1 Type 3 neovascularization with dilated choroid showing poor response to anti-vascular endothelial growth factor therapy, thus suggesting an active role of choroid in Type 3 lesions.
UR - http://www.scopus.com/inward/record.url?scp=84994087068&partnerID=8YFLogxK
UR - http://www.scopus.com/inward/citedby.url?scp=84994087068&partnerID=8YFLogxK
U2 - 10.3928/23258160-20161004-10
DO - 10.3928/23258160-20161004-10
M3 - Article
VL - 47
SP - 956
EP - 959
JO - Ophthalmic Surgery Lasers and Imaging Retina
JF - Ophthalmic Surgery Lasers and Imaging Retina
SN - 2325-8160
IS - 10
ER -