Abstract
A case is reported of a man, aged 67 years, in a toxi infectious condition resulting from gall bladder necrosis due to thrombosis of the cystic artery, who also suffered from secondary metabolic decompensation aggravated by pro existent renal failure, and who had three generalized convulsive attacks. A few hours later, the patient was limp, drowsy and somewhat confused; the EEG showed a slow dysrhythmia which responded only partially to sensitive sensory stimulation, and a right frontal irritation focus. Intravenous administration of diazepam induced clinical sleep and also caused the appearance of suppression bursts lasting 3 to 4 seconds and recurring every 8 minutes.
| Original language | Italian |
|---|---|
| Pages (from-to) | 216-225 |
| Number of pages | 10 |
| Journal | Rivista di Neurologia |
| Volume | 45 |
| Issue number | 2 |
| Publication status | Published - 1975 |
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ASJC Scopus subject areas
- Clinical Neurology
Cite this
SCOPPI SOPPRESSORI DOPO DIAZEPAM I.V. / Smirne, S.; Menegati, E.; Comi, G.; Passerini, D.
In: Rivista di Neurologia, Vol. 45, No. 2, 1975, p. 216-225.Research output: Contribution to journal › Article
}
TY - JOUR
T1 - SCOPPI SOPPRESSORI DOPO DIAZEPAM I.V.
AU - Smirne, S.
AU - Menegati, E.
AU - Comi, G.
AU - Passerini, D.
PY - 1975
Y1 - 1975
N2 - A case is reported of a man, aged 67 years, in a toxi infectious condition resulting from gall bladder necrosis due to thrombosis of the cystic artery, who also suffered from secondary metabolic decompensation aggravated by pro existent renal failure, and who had three generalized convulsive attacks. A few hours later, the patient was limp, drowsy and somewhat confused; the EEG showed a slow dysrhythmia which responded only partially to sensitive sensory stimulation, and a right frontal irritation focus. Intravenous administration of diazepam induced clinical sleep and also caused the appearance of suppression bursts lasting 3 to 4 seconds and recurring every 8 minutes.
AB - A case is reported of a man, aged 67 years, in a toxi infectious condition resulting from gall bladder necrosis due to thrombosis of the cystic artery, who also suffered from secondary metabolic decompensation aggravated by pro existent renal failure, and who had three generalized convulsive attacks. A few hours later, the patient was limp, drowsy and somewhat confused; the EEG showed a slow dysrhythmia which responded only partially to sensitive sensory stimulation, and a right frontal irritation focus. Intravenous administration of diazepam induced clinical sleep and also caused the appearance of suppression bursts lasting 3 to 4 seconds and recurring every 8 minutes.
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UR - http://www.scopus.com/inward/citedby.url?scp=0016588064&partnerID=8YFLogxK
M3 - Articolo
VL - 45
SP - 216
EP - 225
JO - Rivista di Neurologia
JF - Rivista di Neurologia
SN - 0035-6344
IS - 2
ER -